Herpes simplex
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An den 2022 Stiftung Warentest Resultater aus Däitschland war d'Zefriddenheet vum Konsument mam ModelDerm nëmme liicht manner wéi mat bezuelte Telemedizin Konsultatiounen.
An den 2022 Stiftung Warentest Resultater aus Däitschland war d'Zefriddenheet vum Konsument mam ModelDerm nëmme liicht manner wéi mat bezuelte Telemedizin Konsultatiounen.
Herpes simplex op der Lipp.
Herpes simplex – d'Herpes Simplex Infektioun an de Fanger ass méi heefeg bei jonke Kanner wéi bei Erwuessenen.
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ReferencesHSV-1-Infektioun progresséiert duerch eng primär Infektioun vun Epithelzellen, gefollegt vun enger Latenzphase, haaptsächlech an Neuronen, an eng Reaktivéierung. HSV-1 verursaacht allgemeng initial an widderhuelend vesikulär Ausbréch, haaptsächlech am Mond an an der Genitalmucosa. Seng Manifestatiounen reichen vun orolabial Herpes bis zu verschiddene Konditiounen wéi herpetesch Folliculitis, Hautinfektiounen, okulär Bedeelegung, an schwéier Fäll wéi Herpes Ensephalitis. Antivirale Therapie hëlleft, d'HSV-Infektioun ze managen.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 1 (HSV-1) is a member of the Alphaherpesviridae subfamily. Its structure is composed of linear dsDNA, an icosahedral capsid that is 100 to 110 nm in diameter, with a spikey envelope. In general, the pathogenesis of HSV-1 infection follows a cycle of primary infection of epithelial cells, latency primarily in neurons, and reactivation. HSV-1 is responsible for establishing primary and recurrent vesicular eruptions, primarily in the orolabial and genital mucosa. HSV-1 infection has a wide variety of presentations, including orolabial herpes, herpetic sycosis (HSV folliculitis), herpes gladiatorum, herpetic whitlow, ocular HSV infection, herpes encephalitis, Kaposi varicelliform eruption (eczema herpeticum), and severe or chronic HSV infection. Antiviral therapy limits the course of HSV infection.
Herpes simplex virus type 2 (HSV-2) ass eng verbreet Infektioun, déi ongeféier 22 % vun deenen Erwuessenen ab 12 Joer a méi beaflosst, am Ganzen 45 Milliounen Erwuessenen an den USA. Wärend HSV-1 typesch mëndlech Wunden verursaacht, kann et och zu Genitalläsiounen féieren. Wéi och ëmmer, wann d'Patienten genital Läsionen hunn, ass HSV-2 normalerweis d'Haaptsuerg. D'Symptomer vun engem HSV-2 Ausbroch sinn dacks vage, wéi genital Jucken an Reizung, wat d'Diagnostik an d'Behandlung verspéit. Dës Verzögerung kann zu enger weiderer Iwwerdroung un oninfizéiert Leit féieren.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus type 2 (HSV-2) continues to be a common infection, affecting approximately 22% of adults ages 12 and older, representing 45 million adults in the United States alone. While HSV-1 often affects the perioral region and can be known to cause genital lesions, HSV-2 is more commonly the consideration when patients present with genital lesions. Despite this, most outbreaks of the infection will present with nonspecific symptoms such as genital itching, irritation, and excoriations, which may cause diagnosis and treatment to be delayed. As a result, further exposure to uninfected individuals may occur.
Herpes simplex virus (HSV) verursaacht allgemeng Infektiounen wéi genital Herpes an Erkältungen bei Teenagern an Erwuessenen. Wann HSV e Puppelchen an den éischte 4–6 Wochen vum Liewen infizéiert, kann et zu enger schwéierer Krankheet mat éischte Konsequenzen féieren. Eng séier Diagnostik vun neonataler HSV‑Infektioun ass entscheedend, fir d'Krankheet ze stoppen, eng Verschlechterung ze verhënneren an neurologesch Komplikatiounen ze vermeiden (och Doud).
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex virus (HSV), a member of the Herpesviridae family, is a well-known cause of infections including genital herpes and herpes labialis in the adolescent and adult population. Transmission of HSV infection to an infant during the first 4-6 weeks of life can lead to devastating disease with the potential for poor outcomes. Early diagnosis is imperative when evaluating neonatal HSV infection in order to prevent further disease progression, neurological complications, and even death.
Herpes simplex Infektioun ass ganz heefeg a ka vu schwangere Fraen op hir Puppelcher iwwerginn. Dëse Virus kann seriéis Gesondheetsprobleemer oder souguer Doud bei Neigebueren verursaachen. Obwuel et selten während der Schwangerschaft selwer ass, geschitt et dacks während der Gebuert. De Risiko ass héchst, wann d'Mamm an de spéidere Stadien vun der Schwangerschaft infizéiert gëtt. Allerdéngs kann dëse Risiko reduzéiert ginn, andeems Dir antiviral Medikamenter benotzt oder a bestëmmten Situatiounen eng C‑Sektioun auswielt.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Infection with herpes simplex is one of the most common sexually transmitted infections. Because the infection is common in women of reproductive age it can be contracted and transmitted to the fetus during pregnancy and the newborn. Herpes simplex virus is an important cause of neonatal infection, which can lead to death or long-term disabilities. Rarely in the uterus, it occurs frequently during the transmission delivery. The greatest risk of transmission to the fetus and the newborn occurs in case of an initial maternal infection contracted in the second half of pregnancy. The risk of transmission of maternal-fetal-neonatal herpes simplex can be decreased by performing a treatment with antiviral drugs or resorting to a caesarean section in some specific cases.
Herpes simplex virus (HSV) Typen 1 an 2 infizéieren vill Leit weltwäit. Normalerweis bleift de Virus roueg an den Nervenzellen nodeems et d’Haut infizéiert huet, awer et kann spéider reaktivéieren a kale Geschwëster verursaachen. Heiansdo féiert et zu schwéiereren Problemer wéi Aeninfektiounen, Gehirnentzündung oder geféierlech Konditiounen bei Neigebueren an Leit mat schwaachem Immunsystem. Wärend déi aktuell Medikamenter hëllefen, Infektiounen ze kontrolléieren, bleift de Risiko vu Medikamentenresistenz an Nebenwirkungen eng Suerg. Mir brauchen nei Medikamenter, fir de Virus besser ze bekämpfen.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Infection with herpes simplex virus (HSV) types 1 and 2 is ubiquitous in the human population. Most commonly, virus replication is limited to the epithelia and establishes latency in enervating sensory neurons, reactivating periodically to produce localized recurrent lesions. However, these viruses can also cause severe disease such as recurrent keratitis leading potentially to blindness, as well as encephalitis, and systemic disease in neonates and immunocompromised patients. Although antiviral therapy has allowed continual and substantial improvement in the management of both primary and recurrent infections, resistance to currently available drugs and long-term toxicity pose a current and future threat that should be addressed through the development of new antiviral compounds directed against new targets.
Et ginn zwou Zorte vum Herpes simplex Virus, Typ 1 (HSV‑1) an Typ 2 (HSV‑2). HSV‑1 verursaacht méi heefeg Infektiounen ronderëm de Mond, während HSV‑2 méi heefeg genital Infektiounen verursaacht. Si ginn duerch direkte Kontakt mat der infizéierter Persoun iwwerdroen. Genital Herpes gëtt als sexuell iwwerdroen Infektioun klasséiert. Et kann op e Pëppchen während der Gebuert verbreet ginn. No der Infektioun ginn d'Viren laanscht sensorësch Nerven an den Nervenzellkierper transportéiert, wou se liewenslaang wunnen. Ursaachen vun der Wiederhuelung kënnen Ofsenkung vun der Immunfunktioun, Stress a Sonneliichtbelaaschtung enthalen.
Am meeschte Fäll ginn antivirale Medikamenter nëmmen geholl, wann d'Symptomer schwéier sinn. Antivirale Medikamenter kënnen all Dag geholl ginn, fir Leit mat widderhuelende Infektiounen. Et gëtt keng Impfung verfügbar, an d'Shingles‑Impfung verhënnert net Herpes simplex. Behandlungen mat antiviralen Medikamenter wéi Aciclovir oder Valaciclovir kënnen d'Gravitéit vun de Symptomer reduzéieren.
Weltwäit Prävalenz vun HSV‑1 oder HSV‑2 ass tëscht 60 % an 95 % bei Erwuessenen. HSV‑1 ass normalerweis während der Kandheet erwannt. Eng geschätzte 536 Mio. Leit weltwäit (16 % vun der Bevëlkerung) waren mat HSV‑2 infizéiert, mat héicherer Prävalenz bei Fraen an deenen an den Entwécklungslänner. Déi meescht Leit mat HSV‑2 realiséieren net, datt si infizéiert sinn.
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